Dr. Melinda Connor’s Dramatic Findings on X39
Fast Facts on X39 Studies
By Dr. Melinda H. Connor
The Metabolism Pilot
- 17 Amino acid changes at significance at the end of the week.
- Produced anti-inflammatory effects based on amino acid results.
- A reduction in blood pressure at significance.
- Improves quality of sleep.
- Increased vitality.
- Improved short term memory.
The Metabolism Double Blind
- Increase in 8 amino acids at significant levels over the 3 time periods.
- Increase in 3 amino acids at near significance.
- Improved short term memory at significant levels over 7 days.
- Improved mid term memory at near significance in 7 days.
- Improved quality of sleep at significant levels within 24 hours.
- Increase in Vitality at near significant levels in 7 days.
- Reduction in blood pressure at significance in 7 days.
- Improved muscle relaxation at near significance in 7 days.
- Improved Heart Rate Variability at significance in 7 days.
- Increase in depth of respiration at near significance in 7 days. (Breathing more deeply.) The Blood Plasma Pilot
- Increased production at significance of GHK in 24 hours.
- Increased production at significance of GHK-Cu in 7 days.
Research GHK-Cu Copper Peptide
Dr Melinda Connor spoke at the Lifewave Nova Conference Feb 15, 2021 about her lastest research on X39.
Regenerative and Protective Actions of the GHK-Cu Peptide in the Light of the New Gene Data
Loren Pickart and Anna Margolina*

Published online 2018 Jul 7. doi: 10.3390/ijms19071987
PMCID: PMC6073405
PMID: 29986520
This article has been cited by other articles in PMC
Abstract
The human peptide GHK (glycyl-l-histidyl-l-lysine) has multiple biological actions, all of which, according to our current knowledge, appear to be health positive. It stimulates blood vessel and nerve outgrowth, increases collagen, elastin, and glycosaminoglycan synthesis, as well as supports the function of dermal fibroblasts. GHK’s ability to improve tissue repair has been demonstrated for skin, lung connective tissue, boney tissue, liver, and stomach lining. GHK has also been found to possess powerful cell protective actions, such as multiple anti-cancer activities and anti-inflammatory actions, lung protection and restoration of chronic obstructive pulmonary disease (COPD) fibroblasts, suppression of molecules thought to accelerate the diseases of aging such as NFκB, anti-anxiety, anti-pain and anti-aggression activities, DNA repair, and activation of cell cleansing via the proteasome system. Recent genetic data may explain such diverse protective and healing actions of one molecule, revealing multiple biochemical pathways regulated by GHK.
Keywords: GHK, GHK-Cu, gene profiling, wound healing, COPD, skin regeneration, anti-oxidant, fibrinogen
Loren Pickart, Jessica Michelle Vasquez-Soltero, Anna Margolina
Oxid Med Cell Longev. 2012; 2012: 324832. Published online 2012 May 10. doi: 10.1155/2012/324832PMCID: PMC3359723
Article PubReader PDF–531K Cite

Biomed Res Int. 2014; 2014: 151479.
Published online 2014 Sep 11. doi: 10.1155/2014/151479
PMCID: PMC4180391
PMID: 25302294
GHK and DNA: Resetting the Human Genome to Health
Loren Pickart,*Jessica Michelle Vasquez-Soltero, and Anna Margolina
Author informationArticle notesCopyright and License informationDisclaimer
This article has been cited by other articles in PMC.
Abstract
During human aging there is an increase in the activity of inflammatory, cancer promoting, and tissue destructive genes plus a decrease in the activity of regenerative and reparative genes. The human blood tripeptide GHK possesses many positive effects but declines with age. It improves wound healing and tissue regeneration (skin, hair follicles, stomach and intestinal linings, and boney tissue), increases collagen and glycosaminoglycans, stimulates synthesis of decorin, increases angiogenesis, and nerve outgrowth, possesses antioxidant and anti-inflammatory effects, and increases cellular stemness and the secretion of trophic factors by mesenchymal stem cells. Recently, GHK has been found to reset genes of diseased cells from patients with cancer or COPD to a more healthy state. Cancer cells reset their programmed cell death system while COPD patients’ cells shut down tissue destructive genes and stimulate repair and remodeling activities. In this paper, we discuss GHK’s effect on genes that suppress fibrinogen synthesis, the insulin/insulin-like system, and cancer growth plus activation of genes that increase the ubiquitin-proteasome system, DNA repair, antioxidant systems, and healing by the TGF beta superfamily. A variety of methods and dosages to effectively use GHK to reset genes to a healthier state are also discussed.
1. Introduction
According to the Administration on Aging (http://www.aoa.gov/), there were 39 million people aged 65 and older in 2009 which constituted 12% of the American population. By 2030 it is expected that 19% of the population will be over 65. With life expectancy continuing to increase, we may expect that this trend is here to stay. Unfortunately, with advanced age comes not only wisdom but also many age-related pathological conditions that account for the high rates of hospitalization, increased cost of health care and decreased quality of life. Today, more than ever, there is an urgent need to find safe, easy-to-administer, cost-effective methods, which could not only delay the onset of the age related diseases, but also restore health.
It now becomes increasingly clear that the primary cause of human aging and its attendant diseases is changes in the activity of the human genome. During aging there is an increase in the activity of inflammatory, cancer promoting, and tissue destructive genes plus a decrease in the activity of regenerative and reparative genes [1].
The most exciting discovery of the past decades is that these changes in gene activity can be reversed, often by quite simple and natural molecules [2]. Recent discoveries on the actions of the human tripeptide GHK (glycyl-L-histidyl-L-lysine) to reset gene expression of human cells to a more healthy state may open a door to the therapeutic resetting of genes in the elderly. This can be useful as a preventative measure and a complimentary treatment for conditions typically associated with aging such as cancer, Alzheimer’s, chronic obstructive lung disease (COPD), nephropathy, and retinopathy.
